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RQS Scoring Log

Adamax

Nootropics & Neuroprotection· Animal / In Vitro· Scored August 2026· View compound profile →

Adamax is a synthetic nootropic peptide built on the Semax backbone with two pharmacokinetic modifications: N-terminal acetylation and a C-terminal adamantane group. The rationale is sound on paper, but Adamax has never been studied independently in any species. Its RQS score reflects the actual evidence base, not the theoretical one.

11 /100
Insufficient Evidence
No published study of Adamax exists in any species. All proposed mechanisms are inferences from parent compounds Semax and P21. The score reflects the actual evidence base only.
7-Dimension Breakdown
DimensionMaxNotesScore
Study Design25 No published study of Adamax exists in any species — not in vitro, not animal, not human. Scored 2 rather than 0 to reflect the well-characterized preclinical evidence for the parent compound Semax, from which structural inferences are made. 2 / 25
Sample Size20 No human participants. No animal participants in any study of Adamax itself. 0 / 20
Replication20 Zero replication. There is nothing to replicate. No single published study of the compound exists under its own name in any database. 0 / 20
Journal Impact Factor15 No peer-reviewed journal publication on Adamax. The 2 points reflect low-IF Russian pharmacology journals where Semax parent research appears -- a generous proxy given Adamax itself has no journal record. 2 / 15
Funding Independence10 The lineage is Russian academic (Institute of Molecular Genetics). However, the compound now circulates almost exclusively through gray-market research chemical vendors. No formal research funding exists to assess. Partial credit for the academic origin of the parent compound program. 4 / 10
Population Diversity5 No human data. Not applicable. 0 / 5
Researcher h-Index5 Credited to the Semax research lineage (Institute of Molecular Genetics, Russian Academy of Sciences). The researchers behind the parent program have credible academic profiles. Adamax itself has no identifiable primary researcher. 3 / 5
Total10011 / 100
Community Signal

Anecdotal reports in nootropic communities describe Adamax as producing stronger and longer-lasting cognitive effects than Semax, with users noting enhanced focus, improved verbal fluency, and extended duration per dose. These reports are entirely unverifiable and cannot be distinguished from placebo response. PeptideClear does not validate or weight anecdotal claims; they are surfaced here as community signal only.

Scoring Notes
No Independent Research Exists
A PubMed search for "Adamax peptide" returns no results pertaining to this compound. The only PubMed hit for "Adamax" is a machine-learning paper referring to the Adamax optimization algorithm. Every mechanism attributed to Adamax -- BDNF upregulation, TrkB activation, extended half-life, superior BBB penetration -- is structural inference from parent compounds Semax and P21, not measured data. This is acknowledged transparently by the more credible vendor literature. The RQS score of 11 is not a verdict on the compound's potential; it is an accurate reflection of the current evidence base.
Russian Literature Note
The parent compound Semax has a 30+ year clinical history in Russia, documented in Russian pharmacology journals. Those journals score lower on Western impact factor metrics, not because the science is necessarily weaker, but because of publishing system differences. This context applies to the Semax lineage. It does not apply to Adamax, which has no published literature in any language or journal.